When your knee hurts, getting relief is all that's on your mind. Getting the right relief, though, depends on knowing what's wrong. The accurate pathology will lead to the accurate treatment.
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The knee is the largest joint in the body. It's also one of the most complicated. The knee joint is made up of four bones that are linked by muscles, ligaments, and tendons. The femur (large thigh bone) interacts with the two shin bones, the tibia (the larger one) placed towards the inside and the fibula (the smaller one) placed towards the outside. Where the femur meets the tibia is termed the joint line. The patella, (the knee cap) is the bone that sits in the front of the knee. It slides up and down in a groove in the lower part of the femur (the femoral groove) as the knee bends and straightens.
Ligaments are the strong rope-like structures that help join together bones and provide stability. In the knee, there are four major ligaments. On the inner (medial) aspect of the knee is the medial collateral ligament (Mcl) and on the outer (lateral) aspect of the knee is the lateral collateral ligament (Lcl). The other two main ligaments are found in the town of the knee. These ligaments are called the prior cruciate ligament (Acl) and the posterior cruciate ligament (Pcl). They are called cruciate ligaments because the Acl crosses in front of the Pcl. Other smaller ligaments help hold the patella in place in the town of the femoral groove.
Two structures called menisci sit in the middle of the femur and the tibia. These structures act as cushions or shock absorbers. They also help provide stability for the knee. The menisci are made of a tough material called fibrocartilage. There is a medial meniscus and a lateral meniscus. When whether meniscus is damaged it is called a "torn cartilage".
There is other type of cartilage in the knee called hyaline cartilage. This cartilage is a smooth shiny material that covers the bones in the knee joint. In the knee, hyaline cartilage covers the ends of the femur, the femoral groove, the top of the tibia and the underside of the patella. Hyaline cartilage allows the knee bones to move for real as the knee bends and straightens.
Tendons join together muscles to bone. The large quadriceps muscles on the front of the thigh attach to the top of the patella via the quadriceps tendon. This tendon inserts on the patella and then continues down to form the rope-like patellar tendon. The patellar tendon in turn, attaches to the front of the tibia. The hamstring muscles on the back of the thigh attach to the tibia at the back of the knee. The quadriceps muscles are the muscles that straighten the knee. The hamstring muscles are the main muscles that bend the knee.
Bursae are small fluid filled sacs that decrease the disagreement in the middle of two tissues. Bursae also safe bony structures. There are many dissimilar bursae around the knee but the ones that are most prominent are the prepatellar bursa in front of the knee cap, the infrapatellar bursa just below the kneecap, the anserine bursa, just below the joint line and to the inner side of the tibia, and the semimembranous bursa in the back of the knee. Normally, a bursa has very slight fluid in it but if it becomes annoyed it can fill with fluid and come to be very large.
Is it bursitis... Or tendonitis...or arthritis?
Tendonitis commonly affects whether the quadriceps tendon or patellar tendon. Repetitive jumping or trauma may set off tendonitis. The pain is felt in the front of the knee and there is tenderness as well as swelling spicy the tendon. With patellar tendonitis, the infrapatellar bursa will often be inflamed also. Rehabilitation involves rest, ice, and anti-inflammatory medication. Injections are rarely used. Bodily therapy with ultrasound and iontopheresis may help.
Bursitis pain is common. The prepatellar bursa may come to be inflamed particularly in patients who spend a lot of time on their knees (carpet layers). The bursa will come to be swollen. The major concern here is to make sure the bursa is not infected. The bursa should be aspirated (fluid withdrawn by needle) by a specialist. The fluid should be cultured. If there is no infection, the bursitis may be treated with anti-jnflammatory medicines, ice, and Bodily therapy. Knee pads should be worn to prevent a recurrence once the preliminary bursitis is cleared up.
Anserine bursitis often occurs in overweight habitancy who also have osteoarthritis of the knee. Pain and some swelling is noted in the anserine bursa. Rehabilitation consists of steroid injection, ice, Bodily therapy, and weight loss.
The semimembranous bursa can be affected when a outpatient has fluid in the knee (a knee effusion). The fluid will push backwards and the bursa will come to be filled with fluid and cause a sensation of abundance and tightness in the back of the knee. This is called a Baker's cyst. If the bursa ruptures, the fluid will dissect down into the calf. The danger here is that it may look like a blood clot in the calf. A venogram and ultrasound test will help differentiate a ruptured Baker's cyst from a blood clot. The Baker's cyst is treated with aspiration of the fluid from the knee along with steroid injection, ice, and elevation of the leg.
Knock out knee arthritis... Simple steps you can take!
Younger habitancy who have pain in the front of the knee have what is called patellofemoral syndrome (Pfs). Two major conditions cause Pfs. The first is chondromalacia patella. This is a health where the cartilage on the underside of the knee cap softens and is particularly tasteless in young women. other cause of pain behind the knee cap in younger habitancy may be a patella that doesn't track normally in the femoral groove. For both chondromalacia as well as a poorly tracking patella, extra exercises, taping, and anti-inflammatory medicines may be helpful. If the patellar tracking becomes a principal question despite conservative measures, surgical operation is need.
While many types of arthritis may affect the knee, osteoarthritis is the most common. Osteoarthritis normally affects the joint in the middle of the femur and tibia in the medial (inner) compartment of the knee. Osteoarthritis may also involve the joint in the middle of the femur and tibia on the outer side of the knee as well as the joint in the middle of the femur and patella. Why osteoarthritis develops is still being scrutinized carefully. It seems to consist of a complicated interaction of genetics, mechanical factors, and immune principles involvement. The immune principles attacks the joint straight through a aggregate of degradative enzymes and inflammatory chemical messengers called cytokines.
Patients will sometimes feel a sensation of rubbing or grinding. The knee will come to be stiff if the outpatient sits for any distance of time. With local inflammation, the outpatient may palpate pain at night and get relief from sleeping with a pillow in the middle of the knees. Occasionally, locking and clicking may be noticed. Patients with osteoarthritis may also tear the fibrocartilage cushions (menisci) in the knee more for real than habitancy without osteoarthritis.
So how is the arthritis treated? An inevitable place to start is weight reduction for patients who carry around too many pounds.
Strengthening exercises for the knee are also beneficial for many people. These should be done under the supervision of a physician or Bodily therapist.
Other therapies consist of ice, anti inflammatory medicines, and occasionally steroid injections. Glucosamine and chondroitin supplements may be helpful. A word of caution... Make sure the preparation you buy is pure and contains what the label says it does. The supplement business is unregulated... So buyer beware!
Injections of the knee with viscosupplements - lubricants- are particularly beneficial for many patients. extra braces may help to unload the part of the joint that is affected.
Arthroscopic techniques may be beneficial in extra circumstances. Occasionally, a surgical policy called an osteotomy, where a wedge of bone is removed from the tibia to "even things out," may be recommended. Joint transfer surgical operation is required for end stage knee arthritis.
Research is being done to design medicines that will slow down the rate of cartilage loss. Targets for these new therapies consist of the destructive enzymes and/or cytokines that degrade cartilage. It is hoped that by inhibiting these enzymes and cytokines and by boosting the potential of cartilage to mend itself, that therapies designed to for real reverse osteoarthritis may be created. These are referred to as disease-modifying osteoarthritis drugs or "Dmoads." Genetic markers may identify high risk patients who need more aggressive therapies.
Newer compounds that are injected into the knee and provide curative as well as lubrication are also being developed. And finally, less invasive surgical techniques are also being looked at. new technological advances in "mini" knee transfer look very promising.
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